β1-adrenergic receptor polymorphisms confer differential function and predisposition to heart failure

Mutations in the Gene Encoding for the β 2 -adrenergic Receptor in Normal and Asthmatic Subjects

American Gastroenterological Association Institute Technical Review on the Use of Thiopurines, Methotrexate, and Anti–TNF-α Biologic Drugs for the Induction and Maintenance of

α 2A -Adrenergic Receptor Stimulated Calcium Release Is Transduced by G i -Associated G βγ -Mediated Activation of Phospholipase C †

Serine 232 of the α 2A -Adrenergic Receptor Is a Protein Kinase C-Sensitive Effector Coupling Switch †

Role of the Amino Terminus of the Third Intracellular Loop in Agonist-Promoted Downregulation of the α 2A -Adrenergic Receptor †

Altering the Receptor−Effector Ratio by Transgenic Overexpression of Type V Adenylyl Cyclase:  Enhanced Basal Catalytic Activity and Function without Increased Cardiomyocyte β-Adrenergic

Reduction of (η 5 -C 5 Ph 4 q)Ru(CO) 2 Br (q = 2,5-Benzoquinonyl):  Evidence for an 18 − δ Metalate Anion with a Reduced (Semiquinone) Substituent

Fusion of β 2 -Adrenergic Receptor to G αs in Mammalian Cells:  Identification of a Specific Signal Transduction Species Not Characteristic of Constitutive Activation or Precoupling

Direct On-Resin Synthesis of Peptide- α Thiophenylesters for Use in Native Chemical Ligation

Agonist-Mediated Downregulation of G αi via the α 2 -Adrenergic Receptor Is Targeted by Receptor-G i Interaction and Is Independent of Receptor Signaling and Regulation †

α 2A - and α 2C -Adrenergic Receptors Form Homo- and Heterodimers:  The Heterodimeric State Impairs Agonist-Promoted GRK Phosphorylation and β-Arrestin Recruitment †

Catalytic Cyclooligomerization of Thietane by Dirhenium Carbonyl Complexes.

Functional Receptor Coupling to G i Is A Mechanism of Agonist-Promoted Desensitization of the β 2 -Adrenergic Receptor